modification function
HDLdebugger: Streamlining HDL debugging with Large Language Models
Yao, Xufeng, Li, Haoyang, Chan, Tsz Ho, Xiao, Wenyi, Yuan, Mingxuan, Huang, Yu, Chen, Lei, Yu, Bei
In the domain of chip design, Hardware Description Languages (HDLs) play a pivotal role. However, due to the complex syntax of HDLs and the limited availability of online resources, debugging HDL codes remains a difficult and time-intensive task, even for seasoned engineers. Consequently, there is a pressing need to develop automated HDL code debugging models, which can alleviate the burden on hardware engineers. Despite the strong capabilities of Large Language Models (LLMs) in generating, completing, and debugging software code, their utilization in the specialized field of HDL debugging has been limited and, to date, has not yielded satisfactory results. In this paper, we propose an LLM-assisted HDL debugging framework, namely HDLdebugger, which consists of HDL debugging data generation via a reverse engineering approach, a search engine for retrieval-augmented generation, and a retrieval-augmented LLM fine-tuning approach. Through the integration of these components, HDLdebugger can automate and streamline HDL debugging for chip design. Our comprehensive experiments, conducted on an HDL code dataset sourced from Huawei, reveal that HDLdebugger outperforms 13 cutting-edge LLM baselines, displaying exceptional effectiveness in HDL code debugging.
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Inferring spike-timing-dependent plasticity from spike train data
Synaptic plasticity underlies learning and is thus central for development, memory, and recovery from injury. However, it is often difficult to detect changes in synaptic strength in vivo, since intracellular recordings are experimentally challenging. Here we present two methods aimed at inferring changes in the coupling between pairs of neurons from extracellularly recorded spike trains. First, using a generalized bilinear model with Poisson output we estimate time-varying coupling assuming that all changes are spike-timing-dependent. This approach allows model-based estimation of STDP modification functions from pairs of spike trains. Then, using recursive point-process adaptive filtering methods we estimate more general variation in coupling strength over time. Using simulations of neurons undergoing spike-timing dependent modification, we show that the true modification function can be recovered. Using multi-electrode data from motor cortex we then illustrate the use of this technique on in vivo data.
- Research Report > New Finding (0.70)
- Research Report > Experimental Study (0.46)
QFA2SR: Query-Free Adversarial Transfer Attacks to Speaker Recognition Systems
Chen, Guangke, Zhang, Yedi, Zhao, Zhe, Song, Fu
Current adversarial attacks against speaker recognition systems (SRSs) require either white-box access or heavy black-box queries to the target SRS, thus still falling behind practical attacks against proprietary commercial APIs and voice-controlled devices. To fill this gap, we propose QFA2SR, an effective and imperceptible query-free black-box attack, by leveraging the transferability of adversarial voices. To improve transferability, we present three novel methods, tailored loss functions, SRS ensemble, and time-freq corrosion. The first one tailors loss functions to different attack scenarios. The latter two augment surrogate SRSs in two different ways. SRS ensemble combines diverse surrogate SRSs with new strategies, amenable to the unique scoring characteristics of SRSs. Time-freq corrosion augments surrogate SRSs by incorporating well-designed time-/frequency-domain modification functions, which simulate and approximate the decision boundary of the target SRS and distortions introduced during over-the-air attacks. QFA2SR boosts the targeted transferability by 20.9%-70.7% on four popular commercial APIs (Microsoft Azure, iFlytek, Jingdong, and TalentedSoft), significantly outperforming existing attacks in query-free setting, with negligible effect on the imperceptibility. QFA2SR is also highly effective when launched over the air against three wide-spread voice assistants (Google Assistant, Apple Siri, and TMall Genie) with 60%, 46%, and 70% targeted transferability, respectively.
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Inferring spike-timing-dependent plasticity from spike train data
Stevenson, Ian, Koerding, Konrad
Synaptic plasticity underlies learning and is thus central for development, memory, and recovery from injury. However, it is often difficult to detect changes in synaptic strength in vivo, since intracellular recordings are experimentally challenging. Here we present two methods aimed at inferring changes in the coupling between pairs of neurons from extracellularly recorded spike trains. First, using a generalized bilinear model with Poisson output we estimate time-varying coupling assuming that all changes are spike-timing-dependent. This approach allows model-based estimation of STDP modification functions from pairs of spike trains.
Inferring spike-timing-dependent plasticity from spike train data
Stevenson, Ian, Koerding, Konrad
Synaptic plasticity underlies learning and is thus central for development, memory, and recovery from injury. However, it is often difficult to detect changes in synaptic strength in vivo, since intracellular recordings are experimentally challenging. Here we present two methods aimed at inferring changes in the coupling between pairs of neurons from extracellularly recorded spike trains. First, using a generalized bilinear model with Poisson output we estimate time-varying coupling assuming that all changes are spike-timing-dependent. This approach allows model-based estimation of STDP modification functions from pairs of spike trains. Then, using recursive point-process adaptive filtering methods we estimate more general variation in coupling strength over time. Using simulations of neurons undergoing spike-timing dependent modification, we show that the true modification function can be recovered. Using multi-electrode data from motor cortex we then illustrate the use of this technique on in vivo data.
- Research Report > New Finding (0.70)
- Research Report > Experimental Study (0.46)
- Health & Medicine > Therapeutic Area > Neurology (0.48)
- Law > Statutes (0.40)
- Health & Medicine > Therapeutic Area > Neurology (0.48)
- Law > Statutes (0.40)
- Health & Medicine > Therapeutic Area > Neurology (0.48)
- Law > Statutes (0.40)